miR-UL112-3p promoted glioblastoma cell proliferation, clone formation, migration, and invasion by directly regulating tumor suppressor candidate 3 (TUSC3), and the miR-UL112-3p expression was positively associated with glioma size, differentiation, WHO stage and the overall and disease-free survival of patients (Liang et al., 2017). Here, TUSC3 is linked to glioma.