BCL2L11 and chronic obstructive pulmonary disease: Chen et al. (Chen et al., 2021b) investigated the function of TUG1 in an in vitro COPD model with CSE-treated HPMECs and showed that overexpression led to apoptosis in HPMECs by modulating the miR-9a-5p/Bcl-2-like protein 11 (BCL2L11) axis, suggesting that TUG1/miR-9a-5p/BCL2L11 is a potential effective target for COPD treatment.