CD8A and tuberculosis: Thus, the above results obtained by direct ICS without in vitro antigen stimulation suggest that ZOL/IL2 activation/expansion of Vγ2Vδ2 T cells led to the enhanced ability of αβ CD4+ and CD8+ T cells to constitutively produce anti-TB cytokines and that some αβ T-effector populations trafficked to airway at weeks 3 and 14 after MDR-TB infection.