ACE2 and chronic obstructive pulmonary disease: In addition to the thyroid gland, cardiomyocytes and cardiac pericytes also express ACE-2, which may act as a target for SARS-CoV-2.[11,12] The high inflammatory burden caused by cytokine release in COVID-19 has the potential to induce acute myocardial injury and arrhythmia.[13] Therefore, prompt diagnosis and management of cardiac arrhythmias are important considerations in clinical practice, when dealing with COVID-19 infection.[14] The presence of hypothyroidism might worsen the prognosis in such patients after they are diagnosed with COVID-19.[15–17]