Further, the expression of MIP-1α and MCP-1 by macrophages, T cells, monocytes, and other inflammatory cells enhanced the release of IL-6 or IL-8, which contributed to the pathogenic process of bronchiectasis (Barnes, 2008; Rui Jin and Miao, 2017; Yibing Niu, 2018). This evidence concerns the gene CCL3 and bronchiectasis.