In the severe inflammatory stage after the infection, the EPCR bound in the membrane is damaged and becomes a soluble form due to the delamination from the membrane.[19] Here, we found that the PTMT‐T cells can prevent the damage of membrane‐bound EPCR in neutrophil‐included PBMCs and human umbilical vein endothelial cell (HUVEC). Here, PROCR is linked to infection.