A growing number of cancer-associated proteins have been reported to have the ability to undergo LLPS and form biomolecular condensates, such as speckle-type POZ protein, which is involved in oncogenic substrate degradation [150]; p53-binding protein 1 (53BP1) and FET proteins, which are involved in the DNA damage response and genomic stability [151, 152]; and EWS-FLI1, β-catenin, YAP, and PDZ-binding motif (TAZ), which are involved in transcriptional regulation [114, 153–155]. The gene discussed is TP53BP1; the disease is cancer.