A recent study by You et al., demonstrated that dihydroartemisinin exerted therapeutic effects against bleomycin-induced pulmonary inflammation and secondary pulmonary fibrosis (PF) by inhibiting activated Janus kinase 2 (JAK2), a signal transducer and activator of transcription 3 (STAT3) expression. Here, JAK2 is linked to pemphigus foliaceus.