Our previous publication indeed revealed that the metastatic ovarian cancer cells underwent metabolic reprogramming to utilize lipid metabolism for tumor progression in the fatty acid-enriched microenvironment of the peritoneal cavity through the TAK1/NF-κB signaling [23], evincing particular subsets of ovarian cancer cells might be susceptible toward approaches targeting lipid metabolism. Here, NFKB1 is linked to ovarian carcinoma.