RAB42 and glioblastoma: Then, 35 signaling pathways were observed to be activated in high RAB42 expression GBM patients and the six most significantly activated pathways were SYSTEMIC LUPUS ERYTHEMATOSUS, AUTOIMMUNE THYROID DISEASE, ALLOGRAFT REJECTION, ANTIGEN PROCESSING AND PRESENTATION, P53 SIGNALING PATHWAY, and GLYCOSAMINOGLYCAN DEGRADATION.