A tendency for the increase in the production of IL-1β was detected 6 h post infection (52.29 pg/mg ± 6.05), which continued to reaching a significant peak at 24 h post infection (81.06 pg/mg ± 19.43, p = 0.0315) as compared to non-infected sham rats (32.03 pg/mg ± 2.54) (F(2,12) = 4.332, p = 0.0384). This evidence concerns the gene IL1B and infection.