LGALS3 and Myocardial fibrosis: Gal3 binds to a specific beta-galactosidase which is overexpressed by phagocytic macrophages, and thus endorses the proliferation of myofibroblasts with myocardial collagen deposition—leading to the progression of myocardial fibrosis, inflammation, fibrosis and cardiac remodeling [5,6,7,8,9,10].