GAS5 and chronic thromboembolic pulmonary hypertension: Overexpression or knockdown of GAS5 by rats subcutaneously inoculated with PASMCs transfected with GAS5 overexpression vector or shGAS5 vector in vivo, modulated pulmonary artery wall thickening, angiogenesis and autophagy in a chronic thromboembolic pulmonary hypertension (CTEPH) rat model by targeting miR-382-3p [78].