As far as the mechanisms are concerned, IFN-γ secretion and Vδ1 T cell toxicity against MM cells partially occurred via the T cell receptor (TCR) and other molecules (such as NKG2D, CD3, and CD2 receptors; DNAX accessory molecule-1; and intracellular cell adhesion molecule (ICAM)-1). The gene discussed is ICAM1; the disease is Miyoshi myopathy.