Szulzewsky et al. aimed to identify the mechanisms that drive tumorigenesis in multiple YAP fusions, including YAP1–TFE3 (YAP1–TFE3-fused hemangioendothelioma), YAP1–MAMLD1 (supratentorial ependymoma), YAP1–FAM118B (supratentorial ependymoma), and YAP1–SS18 (cervical squamous cell carcinoma and endocervical adenocarcinoma) [76]. This evidence concerns the gene YAP1 and endocervical adenocarcinoma.