SRC and hypertensive disorder: Further arguments might include modulatory effects on the NO system (NO release’s—BPC 157 induced the NO release on its own—particular effect on NOS inhibition (i.e., opposed hypertension and pro-thrombotic effect of L-NAME) and particular effect on NO over-stimulation (i.e., opposed hypotension and anti-thrombotic, pro-bleeding effect of L-arginine) [39,40,41,42,43,44] controlling vasomotor tone and the activation of Src-Caveolin-1-eNOS pathway [48,49]).