TGFB1 and adenomyosis: Still, given that injury-activated SCs can promote EMT and FMT through the paracrine action of TGF-β [78], it is very likely that SCs play a crucial and lasting role in the genesis of EMID-induced adenomyosis via promoting the EMT of both the SC-derived and endometrial epithelial cells that eventually invade into the myometrium.