After exposure for 24 h and 48 h, the increase in the expression levels of the transcription factor NFkB (known to upregulate cytokines and the transcription of cellular adhesion molecules in the beginning of inflammation) and the chaperone HSP70 (a general marker for cellular stress) point towards a delayed response after acute exposure to DEPM [8]; these findings may indicate endothelial dysfunction and cardiovascular disease [8]. Here, NFKB1 is linked to endothelial dysfunction.