The dynamics between LAT1 and ASCT2 in PCa enable glutamine to enter the cytoplasm via ASCT2, glutamine to activate tumor-inducing pathways (i.e., glycolysis, TCA cycle), glutamine to leave the cytoplasm via LAT1, and leucine to enter the cytoplasm via LAT1 [160]. This evidence concerns the gene SLC7A5 and posterior cortical atrophy.