GSEA for GO term investigation revealed that GC patients in the high-risk group are signally different from the low-risk group in the following biological processes: ncRNA processing, positive regulation of cell cycle, TLR signaling pathway, B-cell-mediated immunity regulation of receptor signaling pathway via STAT, regulation of Notch signaling pathway, cell–cell junction assembly, regulation of apoptotic signaling pathway, Notch signaling pathway, developmental cell growth, and regulation of cell–cell adhesion (Figure 7c, Table S8). Here, SOAT1 is linked to gastric cancer.