ADAMTS13 and endothelial dysfunction: Consistent findings contributing to the hypercoagulability include elevated von Willebrand factor secondary to endothelial dysfunction (Goshua et al., 2020); elevated fibrinogen and FVIII, both acute‐phase proteins (Gabay & Kushner, 1999); reduced thrombomodulin (Goshua et al., 2020); and reduced ADAMTS‐13 with altered von Willebrand factor/ADAMTS‐13 ratio (Favaloro et al., 2021).