Mechanistically, these compounds enhanced the chemotaxis and cytotoxicity of γδ T cells through upregulating MICA/B and downregulating the levels of PD‐L1 and PD‐L2 on CAFs and tumour cells; while upregulating CXCR4, CD107a expression and IFN‐γ production in γδ T cells. This evidence concerns the gene MICA and neoplasm.