SLC16A1 and hyperinsulinemic hypoglycemia: Dominant gain-of-function mutations in the promoter region of SLC16A1 provoking the abnormal expression of MCT1 in pancreatic β-cells have been identified in patients suffering from exercise-induced hyperinsulinemic hypoglycemia (MIM#610021), a clinical condition associated with dysregulated insulin secretion in states of low plasma glucose during anaerobic exercise [4].