NR4A1 and infection: To determine if TCR-independent inflammatory signals during infection influence T cell expression of Nur77-GFP, 50,000 αCD3 and αCD28 pre-activated and rested Nur77-GFP OT-I T cells were transferred into mice infected for 10 days with the PRU strain of T. gondii or PRU expressing OVA (T. gondii-OVA).