Finally, Figure 5(i) shows that TRIB3 expression was widely distributed in different immune subtype – wound healing (C1), IFN-gamma dominant (C2), inflammatory (C3), lymphocyte-depleted (C4), immunologically quiet (C5), and TGF-b dominant (C6) – in ccRCC, indicating that TRIB3 has influence on all immune phenotypes. Here, IFNG is linked to nonpapillary renal cell carcinoma.