CRH and neuropathic pain: As CRF neurons project to numerous extra-amygdalar targets involved in the modulation of averse-affective pain behaviors (Beckerman et al., 2013; Pomrenze et al., 2015, 2019; Dedic et al., 2018; de Guglielmo et al., 2019; Neugebauer et al., 2020), we expect that the deactivation of CeA-CRF neurons would correspond with beneficial behavioral effects observed here in a neuropathic pain model.