CNTN1 and congenital bilateral aplasia of vas deferens from CFTR mutation: The current study suggested the LINC00702–miR-181b-5p–SPP1 axis could participate in the development and progression of CAVD and that LINC00702, LINC00092, SPP1, TREM1, TLR8, SDC1, GPM6A, and CNTN1 might be the hub molecules associated with CAVD.