In a Aβ1-40-induced AD model, the expression of inflammation-related genes Aβ, IL-1β, and GFAP were decreased in the hippocampus of rats after ginsenoside Rb1 injection (Lin et al., 2019), indicating that ginsenoside Rb1 can reduce the neuroinflammation in AD. The gene discussed is GFAP; the disease is Alzheimer disease.