Specifically, the association found between the amount of visceral fat and circulating levels of eotaxin on the one hand, and intima-media thickness on the other, could reinforce the hypothesis that IL-17, released by the visceral adipose tissue, induces eotaxin secretion via the smooth muscle cells present in the atheromatosus vessels of patients suffering from obesity-related NAFLD (Tarantino et al., 2014). The gene discussed is IL17A; the disease is obesity due to melanocortin 4 receptor deficiency.