Cao et al. [20] showed that the deletion of ACE2 enhanced hepatic steatosis, oxidative stress, and inflammation in ACE2 knockout mice, and that both proteins, ACE2 and Ang (1–7), ameliorated inflammation, oxidative stress, and hepatic steatosis in FFA-induced HepG2 cells. Here, ANGPT1 is linked to Hepatic steatosis.