Interestingly, it was found that the immune components in the KIRC TME had the most profound effect of KCNN4, with a significant association between the levels of multiple TIC subtypes and KCNN4 expression (Figures 9A–K), and our results denoted that KCNN4 expression level was negatively linked with macrophage M2 in GBM (Figure 9L). This evidence concerns the gene KCNN4 and glioblastoma.