Huang et al., (2017) found that miR-101 was downregulated in the lungs of patients with idiopathic pulmonary fibrosis (IPF), and overexpression of miR-101 in the mouse lungs ameliorated bleomycin-induced lung fibrosis by suppressing the TGF-β-induced activation and WNT5a-induced proliferation of lung fibroblasts. The gene discussed is TGFB1; the disease is pulmonary fibrosis.