The family includes antiapoptotic (Bcl-2 and Bcl-xL) and proapoptotic (Bax, Bid, Bak, and Bcl-x) members; balancing their interaction ensures proper apoptotic regulation and cell fate in response to fetal development signals, tissue homeostasis, tumor surveillance, and cell stresses (6). This evidence concerns the gene BCL2L1 and neoplasm.