CXCL8 and glioblastoma: Hypoxia-induced ALKBH5 in GBM cells cleared m6A deposition of lncRNA NEAT1, stabilized transcripts and facilitated NEAT1-mediated parabasal assembly, which led to relocalization of the transcriptional repressor SFPQ from the promoter of CXCL8 to the parabasal and thereby promoted the expression of CXCL8/IL8 (161).