Increased miR-23 levels are associated with the maintenance of the integrity of endothelial structures (60), and has been proposed to evaluate the presence and severity of coronary lesions in CHD patients (61) while, in an experimental autoinmmune murine model of encephalomyelitis, miR-23b suppresses leukocyte migration and pathogenesis by targeting CCL7 (39). This evidence concerns the gene CCL7 and coronary artery disorder.