T-ALL displayed higher EHMT2/G9a expression compared to other tumor types [16] (Fig. 1D), normal human thymocytes (Fig. 1E, Supplementary Fig. 1G), or lymphocytes (Fig. 1F) independent from the activation of known transcription factor or recurrent mutations (Supplementary Fig. 1H). Here, EHMT2 is linked to neoplasm.