In addition, RRLs such as PKR, MDA5, TLR3, and signaling intermediates, MAVS, TNF receptor‐associated factor 3 (TRAF3), TRAF6, TBK1, and IKKε, were present in SG formed during infection in 20% of infected cells, suggesting that avSGs function as a scaffold for viral RNA recognition by RLRs in wild‐type infection by IBV (Gao et al., 2021). The gene discussed is TBK1; the disease is infection.