For this reason, in the future, our research group will investigate (1) DNMT-1 expression detection in MM; (2) the effects of DNMT-1 overexpression or knockdown on the expression of miR-23, and on the miR-23 promoter’s methylation; and (3) the recruitment of DNMT-1 to the CpG islands in the promoter region of miR-23. The gene discussed is DNMT1; the disease is Miyoshi myopathy.