In addition, EP treatment in mice significantly suppresses streptozotocin‐induced type 1 diabetes (T1D) development, in line with the reduced infiltration of cells into the pancreatic islets, enhanced regulatory T cell (Treg) and suppressed IFN‐γ‐producing cells, leading to preservation of β‐cell functions.56 This evidence concerns the gene IFNG and type 1 diabetes mellitus.