The positive correlations found in our study between plasma AMH and TC-derived androgens, as well as between follicle count and testosteronemia, strengthen the association between PCOS and intrafollicular hyperandrogenism and, at the same time, corroborate literature data supporting the pathogenic role of androgen excess in the development of PCOM [34]. The gene discussed is AMH; the disease is polycystic ovary syndrome.