DNM1L and glioma: Of note, as stated before, the overexpression of Hsp90 and Drp1 dephosphorylation at Ser637 increased erastin-induced lipid peroxidation in glioma cells, but as indicated, this effect was completely reversed by ferroptosis inhibitors (ferrostatin-1), but not by inhibitors of apoptosis (ZVAD- FMK) (Supplementary Fig. 5D–I).