To pinpoint specific lncRNAs that have potential roles in p53-mediated tumor-suppressive functions, we analyzed seven additional RNA-seq datasets where p53 wild-type (p53WT) cells were treated with Nutlin-3, a compound that blocks the binding of p53 to Mdm2 (Vassilev et al., 2004), triggering p53 activation independent of DNA damage, or controls (‘Materials and methods’; Supplementary file 2b). The gene discussed is TP53; the disease is neoplasm.