The study by Emin et al. found that the internalization of a specific protein called CD59 in patients with OSA promoted endothelial inflammation and damage, while atorvastatin treatment stabilized CD59 protein on the endothelial cell surface, protecting them from hypoxia-induced injury and consequently increased cardiovascular risk (Emin et al., 2016). This evidence concerns the gene CD59 and obstructive sleep apnea syndrome.