KCNJ1 and colitis: In addition, the mRNA expression levels of Kcnj1 and Kcnma1, secretory K+ channel genes, were upregulated in the DSS+Veh group when compared with the control group; however, tegoprazan or rabeprazole administration reduced these DSS-induced increased expression levels (Figure 2H), indicating that tegoprazan relieves colitis by suppressing the expression of proinflammatory factors, but not K+ channels.