Considering the current circumstances, we propose (1) re-evaluation of foretinib in the second-line setting for NSCLC patients who acquired resistance to capmatinib or tepotinib by secondary mutations or (2) development of a novel type II MET-TKI with a long tail that interacts with the residue G1163 of the MET. Here, MET is linked to non-small cell lung carcinoma.