SJL mice, similar to BALB/c mice, displayed limited IFN-γ availability in mLN, spleen and siLP (Supplementary Fig. S4a), poor small intestinal Th2/1 accumulation at early stage of infection (Supplementary Fig. S4b) and stronger M2 polarization in the peritoneal cavity compared to susceptible C57BL/6 mice (Supplementary Fig. S4c). The gene discussed is IFNG; the disease is infection.