The enrichment results disclose the pivotal participation of inflammatory response, defense response, apoptotic, cell migration and adhesion, and reactive oxygen species metabolic in the SMYAD against AAA, as well as the collaborative involvement of the TNF signalling pathway, NF-kappa B signalling pathway, PI3K-Akt signalling pathway, and IL-17 signalling pathway [65–69]. This evidence concerns the gene IL17A and triple-A syndrome.