Furthermore, it induced apoptosis in human prostate cancer cells PC-3 and DU-145 at higher concentrations (400–1000 μg/mL) through the regulation of the expression of Bcl2/Bax proteins [56], inhibiting proliferation and apoptosis of human cervical carcinoma (HeLa) cells via the mitochondrial pathway [57]. This evidence concerns the gene BCL2 and prostate cancer.