Liu et al. also reported more hypertension with increases in urine sediment, triglycerides, blood glucose, BUN, serum creatinine, phosphorus, parathyroid hormone, 24-h urinary albumin, 24-h urinary α1-microglobulin, and activity of urine N-acetyl-βD-glucosaminidase (NAG), along with lower eGFR, serum albumin, complement 3, 24-h urinary calcium, urinary volume, and urinary pH in patients with LN and HUA compared to those without HUA [13]. This evidence concerns the gene PTH and hypertensive disorder.