These results suggest that baicalin enhances intestinal repair after injury by promoting Th17 cell differentiation and IL-22 production, consistent with findings from a previous study that baicalin regulates Th1, Th17, and Treg responses to ameliorate sepsis-associated pancreatic injury through the ras homolog family member A -Rho kinase pathway [13]. This evidence concerns the gene IL22 and Sepsis.